Helicobacter pylori

One-Sentence Definition

Helicobacter pylori is a microaerophilic, urease-positive curved Gram-negative bacillus that colonizes the human stomach and drives gastritis, peptic ulcer disease, and risk of gastric adenocarcinoma and MALT lymphoma.

Taxonomy & Morphology

  • Campylobacterales; spiral/curved rods; multiple polar flagella
  • Oxidase-, catalase-, and urease-positive (survives acid via NH₃ from urea)
  • Strictly associated with gastric mucosa in humans (person-to-person / family clustering)

Virulence Highlights

  • Urease — acid resistance
  • CagA (cag PAI — a genomic island) — injected via T4SS; associated with ulcer/cancer risk
  • VacA — vacuolating cytotoxin; allele variants modulate virulence
  • Motility and adhesins for mucus/epithelial niche

Clinical Syndromes

  • Chronic gastritis (nearly universal if colonized)
  • Duodenal/gastric ulcers
  • Increased risk gastric adenocarcinoma; MALT lymphoma (may regress with eradication)
  • Dyspepsia workups — test-and-treat strategies in selected populations

Diagnosis

  • Noninvasive: urea breath test, stool antigen
  • Invasive: biopsy urease, histology, culture (for AST when refractory)
  • Serology: marks exposure; poor for test-of-cure
  • Molecular detection / resistance PCR on stool or biopsy increasingly used

Treatment Notes (conceptual)

  • Multi-drug eradication regimens (PPI + antibiotics); local resistance guides empiric choice
  • Confirm eradication after therapy when clinically indicated
  • Avoid endless empiric courses without testing — stewardship + guideline adherence

AMR

  • Clarithromycin resistance (23S rRNA mutations) — major empiric-regimen failure driver
  • Metronidazole, levofloxacin, amoxicillin, rifabutin resistance patterns vary geographically
  • Culture/AST or molecular resistance testing for refractory disease

Genomics & Computational Notes

  • High within-host diversity; recombination; multilocus / WGS for phylogeography
  • cag PAI presence/absence and VacA genotyping for research risk stratification
  • Example of chronic colonization + cancer association in bacterial pathogenesis teaching

Active Recall

  1. How does urease allow gastric survival?
  2. What is CagA’s delivery system, and why does it matter clinically?
  3. Why is serology a poor test-of-cure?