Colistin Resistance

One-Sentence Definition

Colistin (polymyxin E) resistance is reduced activity of last-line cationic peptide antibiotics against Gram-negatives, mediated mainly by LPS lipid A modifications (chromosomal regulatory mutations or mobile mcr phosphoethanolamine transferases).

Simple Explanation

Colistin punches holes in the outer membrane by sticking to bacterial endotoxin. Resistant bugs chemically remodel that surface charge so colistin can no longer bind well — sometimes via a plasmid gene that can jump between species.

Detailed Scientific Explanation

Mechanism classExamplesMobility
Chromosomal regulatoryPhoPQ, PmrAB, MgrB disruption (Klebsiella) → L-Ara4N / pEtN on lipid AVertical / mutational
Mobile mcrmcr-1… phosphoethanolamine transferasesPlasmid (Horizontal Gene Transfer)
IntrinsicProteus, Serratia, Burkholderia — naturally less susceptibleN/A

Detection challenges:

  • Colistin MIC methods finicky (broth microdilution reference; agar dilution/other methods limited)
  • Heteroresistance common
  • Molecular: mcr PCR / WGS; chromosomal lesions need careful interpretation (Genotype to Phenotype Prediction)

Mechanism

Positive charge of polymyxin attracted to negatively charged lipid A → membrane disruption. Adding positively charged moieties (Ara4N, pEtN) reduces binding → survival.

Clinical Importance

  • Used for XDR CRE / MDR Acinetobacter / Pseudomonas when few options remain — nephro/neurotoxicity limits use
  • Resistance rises under selective pressure; infection control + stewardship essential
  • Mobile mcr is a One Health red flag (livestock ↔ human interface) (One Health)

Research Importance

Diagnostic Relevance

  • Reference BMD for MIC; EUCAST/CLSI rules evolve
  • Report mcr when found — epidemiologic importance even if local therapy differs
  • WGS surveillance pipelines flag mcr + MgrB/PhoPQ/PmrAB hits (AMR Gene Databases)

AMR Relevance

Last-line Gram-negative AMR. Often stacked on CRE plasmids already carrying ESBL / Carbapenemases genes — truly extensively drug-resistant phenotypes.

Learning Aids

Clinical Example

Example

Case: NDM-producing E. coli; colistin MIC 4 µg/mL; WGS shows mcr-1 on an IncX4 plasmid.
Question: Chromosomal PhoPQ mutation or mobile resistance?
Answer: Mobile mcr-1 — IPC and One Health implications beyond this patient; seek alternative active agents.

Active Recall Questions

  1. What LPS change reduces colistin binding?
  2. Why is mcr especially concerning compared with MgrB loss?
  3. Which AST method is the reference for colistin MIC?

Connections